Therapy for Stage IV Non-Small-Cell Lung Cancer With Driver Alterations
Treatment
Summary of All Recommendations
- For recommendations with multiple treatment options of the same evidence quality and strength of recommendation, the decision of which agent to offer should be tailored to each patient incorporating both efficacy and toxicity.
- All biomarkers should be available at the time of decision-making.
Clinical Question 1: What are the most effective first-line treatment options for patients’ status based on the driver alterations:
Epidermal growth factor receptor (EGFR)
Exon 19 deletion, Exon 21 L858R substitution
1.1.
Clinicians should offer osimertinib.
(, , M, S )Qualifying Statement: Although Recommendation 1.1 addresses many patients in the target population, the guideline manuscript presents additional options that may be reasonable, based on the evidence reviewed. In addition, use of osimertinib in patients previously treated with adjuvant tyrosine kinase inhibitors (TKIs) is not reflected in this guideline.
1.1.1.
Clinicians may offer osimertinib with platinum doublet chemotherapy or amivantamab plus lazertinib.
(, , M, W )Qualifying Statement: Although Recommendation 1.1 addresses many patients in the target population, the guideline manuscript presents additional options that may be reasonable, based on the evidence reviewed. In addition, use of osimertinib in patients previously treated with adjuvant tyrosine kinase inhibitors (TKIs) is not reflected in this guideline.
Others
1.2.
1.2.1.
For other activating EGFR alterations, (G719X, L861Q, S768I), clinicians may offer osimertinib or
(, , L , W )Qualifying Statement: Recommendation 1.2, 1.2.1, and 1.2.2 excludes exon 20 insertion alterations, T790M.
1.2.2.
1.3.
For any activating EGFR alteration, regardless of programmed death ligand 1 (PD-L1) expression levels (including exon 20 insertions), single-agent immune checkpoint inhibitors should not be offered as first-line therapy.
(, , M, S )Exon 20 insertions
1.4.
1.5.
If amivantamab is not available, clinicians should offer standard treatment following the non-driver alteration guideline.
(, , M, S )Anaplastic lymphoma kinase (ALK)
1.6.
Clinicians should offer alectinib or brigatinib or lorlatinib.
(, , H , S )1.7.
If alectinib, brigatinib, or lorlatinib are not available, clinicians should offer ceritinib or crizotinib.
(, , H , S )ROS1
1.8.
Clinicians may offer repotrectinib, entrectinib, or crizotinib.
(, , M, S )1.9.
If crizotinib, entrectinib, or repotrectinib are not available or not tolerated, clinicians may offer ceritinib or lorlatinib.
(, , L , W )BRAFV600E
1.10.
1.11.
MET exon 14 skipping mutation
1.12.
1.13.
If capmatinib or tepotinib is not available, clinicians may offer standard first-line therapy following the non-driver alteration guidelines.
(, , L , S )RET rearrangement
1.14.
Clinicians should offer selpercatinib.
(, , H , S )1.15.
1.16.
If selpercatinib or pralsetinib are not available, clinicians may offer standard therapy following the non-driver alteration guideline.
(, , L , W )Neurotrophic tyrosine receptor kinase (NTRK) rearrangement
1.17.
Clinicians may offer entrectinib or larotrectinib.
(, , L , S )1.18.
1.19.
For patients with a poor performance status (PS), tyrosine kinase inhibitor may be offered based on drug access and toxicity profile.
(, , L , W )1.20.
Comprehensive genomic biomarker test results should be available and used to guide treatment.
(, , H , S )Qualifying Statement: PDL-1 IHC alone should not be used to guide treatment decisions.
1.21.
Clinical Question 2: What are the most effective second-line and subsequent treatment options for patients based on the driver alterations:
- Due to development of potentially targetable resistance mechanisms, every effort should be made to assess for presence of new mutation by tissue and/or blood next-generation sequencing (NGS) testing.
- If patients have received all targeted options, or if no targeted options are available, clinicians may offer standard therapy following the non-driver alteration guideline.
EGFR
Exon 19 deletion, Exon 21 L858R substitution
2.1.
For patients that develop EGFR T790M resistance alterations in tumor after first- or second-generation EGFR TKIs, clinicians should offer osimertinib.
(, , H , S )2.2.
For patients who have progressive disease on osimertinib or other EGFR TKIs without emergent T790M or other targetable alterations, clinicians may offer platinum-based chemotherapy with or without amivantamab.
(, , M, S )Qualifying Statement: Patients that do not pursue amivantamab plus chemotherapy may also consider chemotherapy plus bevacizumab if they have adenocarcinoma and bevacizumab is deemed safe.
2.2.1.
For patients who progressed on osimertinib (or other 3rd generation TKI), clinicians may offer amivantamab plus carboplatin and pemetrexed.
(, , M, S )2.2.2.
Others
2.3.
ALK
2.4.
2.5.
For patients who have previously received other ALK inhibitors including alectinib or brigatinib, clinicians may offer lorlatinib.
(, , L , S )ROS1
2.6.
For patients who have previously received crizotinib, entrectinib, lorlatinib, or ceritinib, clinicians may offer repotrectinib.
(, , M, S )2.7.
BRAFV600E
2.8.
2.9.
For patients who have previously received BRAF or MEK targeted therapy, clinicians should offer standard first-line therapy following the non-driver alteration guideline.
(, , L , S )2.10.
For BRAF alterations other than BRAFV600E alterations, clinicians should offer standard therapy following the non-driver alteration guideline.
(, , L , S )MET exon 14 skipping mutation
2.11.
2.12.
RET rearrangement
2.13.
2.14.
If selpercatinib or pralsetinib is not available, clinicians may offer treatment following the non-driver alteration guideline.
(, , L , S )NTRK rearrangement
2.15.
For patients who have not received an NTRK inhibitor, clinicians should offer entrectinib or larotrectinib.
(, , L , S )2.16.
Human epidermal receptor factor 2 (HER2)
2.17.
KRAS G12C
2.18.
2.19.
Clinicians may offer treatment with adagrasib.
(, , L , S )Qualifying Statement: Note that adagrasib and sotorasib are approved for patients who have received prior chemotherapy and/or anti-PD-(L)1 for patients with advanced KRAS G12C mutant NSCLC. In the first-line setting, these patients should be offered standard first-line treatment with immune checkpoint inhibitor therapy and/or chemotherapy following the non-driver alteration guideline.
Recommendation Grading
Overview
Title
Therapy for Stage IV Non-Small-Cell Lung Cancer With Driver Alterations
Authoring Organization
American Society of Clinical Oncology
Publication Month/Year
November 12, 2024
Last Updated Month/Year
November 14, 2024
Supplemental Implementation Tools
Document Type
Guideline
External Publication Status
Published
Country of Publication
US
Target Patient Population
Patients with stage IV non–small cell lung cancer (NSCLC) with driver alterations
Inclusion Criteria
Male, Female, Adult
Health Care Settings
Ambulatory, Home health, Hospital, Outpatient
Intended Users
Nurse, nurse practitioner, physician, physician assistant, social worker
Scope
Treatment, Management
Diseases/Conditions (MeSH)
D002289 - Carcinoma, Non-Small-Cell Lung
Keywords
non-small cell lung cancer, Targeted Therapy, Clinical guidelines, ROS-1 fusions, BRAF V600e mutations, RETfusions, MET exon 14 skipping mutations, NTRK fusions
Source Citation
Source Bazhenova L, Ismaila N, Rous FA, et al. Therapy for Stage IV Non-Small Cell Lung Cancer with Driver Alterations: ASCO Living Guideline, Version 2024.2. J Clin Oncol. 2024 November 12. doi: 10.1200/JCO.24.02133
Owen DH, Ismaila N, Freeman-Daily J, et al. Therapy for Stage IV Non-Small Cell Lung Cancer With Driver Alterations: ASCO Living Guideline, Version 2024.1. J Clin Oncol. 2024 May 30. doi: 10.1200/JCO.24.00762